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Without nucleic acid, bacterial cells cannot undergo cellular division, thus the number of bacterial cells will remain the same, achieving bacteriostasis

Research indicates that HGF activation provides neuroprotective effects in cerebral ischemia, spinal cord injuries, and traumatic brain damage by promoting neuronal survival, stimulating angiogenesis, and reducing apoptosis

The degradation process of tryptophan in KP produces a variety of neuroactive metabolites with amino acid-like structures, such as an N-methyl-D-aspartate (NMDA) receptor agonist quinolinic acid (QA), and a neuroprotective NMDA receptor agonist kynurenic acid (KYNA)

Ser-485 in the kinase C-tail is one of the residues that is responsible for the increased rate of autophosphorylation of GRK4 486V [84]